Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. Alternatively, our capacity to rescue SERT functionality in KI midbrain synaptoneurosomes by the inhibition of FAK indicates elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT synapses. Our information https://englandx986amx7.blogsumer.com/profile